20162020

Research output per year

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Research Output

2020

NOX4 is a major regulator of cord blood-derived endothelial colony-forming cells which promotes postischaemic revascularisation

O'Neill, K. M., Campbell, D. C., Edgar, K. S., Gill, E. K., Moez, A., McLoughlin, K. J., O'Neill, C. L., Dellett, M., Hargey, C. J., Abudalo, R. A., O'Hare, M., Doyle, P., Toh, T., Khoo, J., Wong, J., McCrudden, C. M., Meloni, M., Brunssen, C., Morawietz, H., Yoder, M. C. & 6 others, McDonald, D. M., Watson, C. J., Stitt, A. W., Margariti, A., Medina, R. J. & Grieve, D. J., 01 Feb 2020, In : Cardiovascular Research. 116, 2, p. 393-405 13 p.

Research output: Contribution to journalArticle

Open Access
File
1 Citation (Scopus)
29 Downloads (Pure)
2019

The RNA-binding protein QKI controls alternative splicing in vascular cells, producing an effective model for therapy

Caines, R., Cochrane, A., Kelaini, S., Gonzalez, M. V., Yang, C., Eleftheriadou, M., Moez, A., Stitt, A. W., Zeng, L., Grieve, D. J. & Margariti, A., 15 Aug 2019, In : Journal of Cell Science.

Research output: Contribution to journalArticle

2 Citations (Scopus)
2018

Adverse cardiac remodelling in experimental diabetes is regulated by endothelial Nox4 NADPH oxidase-derived reactive oxygen species

Edgar, K., Gill, E., Patterson, E., Hargey, C., Moez, A. & Grieve, D., 16 Jul 2018.

Research output: Contribution to conferenceAbstract

NADPH oxidase 4 is a major regulator of cord blood-derived endothelial colony-forming cells which promotes postischemic revascularisation

O'Neill, K., Campbell, D., Edgar, K., Moez, A., McLoughlin, K., O'Neill, C., Dellett, M., Hargey, C., Abudalo, R., Gill, E., Doyle, P., Toh, T., Khoo, J., McCrudden, C., Brunssen, C., Morawietz, H., Yoder, M. C., Stitt, A., Margariti, A., Medina, R. & 1 others, Grieve, D., 03 Feb 2018.

Research output: Contribution to conferenceAbstract

2017

INFLUENCE OF NADPH OXIDASES NOX4 ISOFORM ON MOUSE IPS DIFFERENTIATION

Moez, A., O'Neill, K., Margariti, A. & Grieve, D., 14 Jun 2017.

Research output: Contribution to conferenceAbstract