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Measuring a cell's response to stress: the p53 pathway.
D. Paul Harkin
*
, Peter A. Hall
*
Corresponding author for this work
Queen's University Belfast
School of Medicine, Dentistry and Biomedical Sciences
Patrick G Johnston Centre for Cancer Research
Research output
:
Contribution to journal
›
Article
›
peer-review
4
Citations (Scopus)
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Medicine & Life Sciences
p53 Genes
68%
DNA Damage
53%
Genes
49%
Binding Sites
47%
Transcription Factors
46%
Genome
41%
Gene Expression
36%
Critical Pathways
35%
purine
35%
Telomerase
35%
Ribonucleases
34%
Mutation
34%
Tumor Suppressor Genes
30%
Oxidoreductases
28%
Up-Regulation
22%
Technology
20%
Apoptosis
18%
Growth
15%
Incidence
14%
Neoplasms
9%
Agriculture & Biology
stress response
100%
genes
42%
DNA damage
40%
cells
39%
binding sites
35%
transcription factors
31%
tumor suppressor genes
29%
telomerase
28%
mutation
28%
gene expression
24%
ribonucleases
22%
genome
22%
purines
21%
apoptosis
18%
neoplasms
15%
incidence
14%
organisms
11%
methodology
11%
Earth & Environmental Sciences
gene
93%
cell
76%
gene expression
41%
mutation
35%
genome
33%
DNA
29%
apoptosis
23%
loss
18%
repair
17%
fold
12%
organism
11%
analysis
9%