Mesenchymal stromal cells (MSC) anti-microbial effect in acute respiratory distress syndrome (ARDS) is mediated in part by enhanced alveolar macrophage phagocytosis through cell contact-dependent mitochondrial transfer leading to improved macrophage bioenergetics

Megan Jackson; Thomas Morrison; Danny McAuley; Cecilia O'Kane; Michael A. Matthay; Anna Krasnodembskaya

doi:10.1183/13993003.congress-2016.PA939

Mesenchymal stromal cells (MSC) anti-microbial effect in acute respiratory distress syndrome (ARDS) is mediated in part by enhanced alveolar macrophage phagocytosis through cell contact-dependent mitochondrial transfer leading to improved macrophage bioenergetics

Megan Jackson
, Thomas Morrison
, Danny McAuley
, Cecilia O'Kane
, Michael A. Matthay
, Anna Krasnodembskaya

School of Medicine, Dentistry and Biomedical Sciences

Research output: Contribution to journal › Meeting abstract › peer-review

7 Citations (Scopus)

Original language	English
Article number	PA939
Pages (from-to)	1
Number of pages	1
Journal	European Respiratory Journal
Volume	48
Issue number	Supplement 60
DOIs	https://doi.org/10.1183/13993003.congress-2016.PA939
Publication status	Published - 08 Nov 2016

Bibliographical note

Megan Jackson, Thomas Morrison, Danny McAuley, Cecilia O'Kane, Michael Matthay, Anna Krasnodembskaya
European Respiratory Journal 2016 48: PA939; DOI: 10.1183/13993003.congress-2016.PA939

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10.1183/13993003.congress-2016.PA939Licence: Unspecified

Human mesenchymal stromal cell regulation of pulmonary macrophage populations in the Acute Respiratory Distress Syndrome
Morrison, T. (Author), Krasnodembskaya, A. (Supervisor), Kissenpfennig, A. (Supervisor) & O'Kane, C. (Supervisor), Jul 2017
Student thesis: Doctoral Thesis › Doctor of Philosophy

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Jackson, M., Morrison, T., McAuley, D., O'Kane, C., Matthay, M. A., & Krasnodembskaya, A. (2016). Mesenchymal stromal cells (MSC) anti-microbial effect in acute respiratory distress syndrome (ARDS) is mediated in part by enhanced alveolar macrophage phagocytosis through cell contact-dependent mitochondrial transfer leading to improved macrophage bioenergetics. European Respiratory Journal, 48(Supplement 60), 1. Article PA939. https://doi.org/10.1183/13993003.congress-2016.PA939

Jackson, Megan ; Morrison, Thomas ; McAuley, Danny et al. / Mesenchymal stromal cells (MSC) anti-microbial effect in acute respiratory distress syndrome (ARDS) is mediated in part by enhanced alveolar macrophage phagocytosis through cell contact-dependent mitochondrial transfer leading to improved macrophage bioenergetics. In: European Respiratory Journal. 2016 ; Vol. 48, No. Supplement 60. pp. 1.

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title = "Mesenchymal stromal cells (MSC) anti-microbial effect in acute respiratory distress syndrome (ARDS) is mediated in part by enhanced alveolar macrophage phagocytosis through cell contact-dependent mitochondrial transfer leading to improved macrophage bioenergetics",

author = "Megan Jackson and Thomas Morrison and Danny McAuley and Cecilia O'Kane and Matthay, \{Michael A.\} and Anna Krasnodembskaya",

note = "Megan Jackson, Thomas Morrison, Danny McAuley, Cecilia O'Kane, Michael Matthay, Anna Krasnodembskaya European Respiratory Journal 2016 48: PA939; DOI: 10.1183/13993003.congress-2016.PA939 ",

year = "2016",

month = nov,

day = "8",

doi = "10.1183/13993003.congress-2016.PA939",

language = "English",

volume = "48",

pages = "1",

journal = "European Respiratory Journal",

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Jackson, M, Morrison, T, McAuley, D , O'Kane, C, Matthay, MA & Krasnodembskaya, A 2016, 'Mesenchymal stromal cells (MSC) anti-microbial effect in acute respiratory distress syndrome (ARDS) is mediated in part by enhanced alveolar macrophage phagocytosis through cell contact-dependent mitochondrial transfer leading to improved macrophage bioenergetics', European Respiratory Journal, vol. 48, no. Supplement 60, PA939, pp. 1. https://doi.org/10.1183/13993003.congress-2016.PA939

Mesenchymal stromal cells (MSC) anti-microbial effect in acute respiratory distress syndrome (ARDS) is mediated in part by enhanced alveolar macrophage phagocytosis through cell contact-dependent mitochondrial transfer leading to improved macrophage bioenergetics. / Jackson, Megan; Morrison, Thomas; McAuley, Danny et al.
In: European Respiratory Journal, Vol. 48, No. Supplement 60, PA939, 08.11.2016, p. 1.

Research output: Contribution to journal › Meeting abstract › peer-review

TY - JOUR

T1 - Mesenchymal stromal cells (MSC) anti-microbial effect in acute respiratory distress syndrome (ARDS) is mediated in part by enhanced alveolar macrophage phagocytosis through cell contact-dependent mitochondrial transfer leading to improved macrophage bioenergetics

AU - Jackson, Megan

AU - Morrison, Thomas

AU - McAuley, Danny

AU - O'Kane, Cecilia

AU - Matthay, Michael A.

AU - Krasnodembskaya, Anna

N1 - Megan Jackson, Thomas Morrison, Danny McAuley, Cecilia O'Kane, Michael Matthay, Anna Krasnodembskaya European Respiratory Journal 2016 48: PA939; DOI: 10.1183/13993003.congress-2016.PA939

PY - 2016/11/8

Y1 - 2016/11/8

U2 - 10.1183/13993003.congress-2016.PA939

DO - 10.1183/13993003.congress-2016.PA939

M3 - Meeting abstract

SN - 0903-1936

VL - 48

SP - 1

JO - European Respiratory Journal

JF - European Respiratory Journal

IS - Supplement 60

M1 - PA939

ER -

Jackson M, Morrison T, McAuley D , O'Kane C, Matthay MA, Krasnodembskaya A. Mesenchymal stromal cells (MSC) anti-microbial effect in acute respiratory distress syndrome (ARDS) is mediated in part by enhanced alveolar macrophage phagocytosis through cell contact-dependent mitochondrial transfer leading to improved macrophage bioenergetics. European Respiratory Journal. 2016 Nov 8;48(Supplement 60):1. PA939. doi: 10.1183/13993003.congress-2016.PA939

Mesenchymal stromal cells (MSC) anti-microbial effect in acute respiratory distress syndrome (ARDS) is mediated in part by enhanced alveolar macrophage phagocytosis through cell contact-dependent mitochondrial transfer leading to improved macrophage bioenergetics

Bibliographical note

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Student theses

Human mesenchymal stromal cell regulation of pulmonary macrophage populations in the Acute Respiratory Distress Syndrome

Cite this