NLRC3 Restrains Responses to a T

Caroline L. Holley, Rebecca C. Coll, Kate Schroder*

*Corresponding author for this work

Research output: Contribution to journalComment/debate

Abstract

The NOD-like receptor protein NLRC3 attenuates myeloid cell inflammatory responses. In this issue of Immunity, Uchimura et al. (2018) reveal additional T-cell-intrinsic functions for NLRC3 in restricting T cell metabolism, T helper 1 and T helper 17 cell responses, and antiviral and autoimmune responses. The NOD-like receptor protein NLRC3 attenuates myeloid cell inflammatory responses. In this issue of Immunity, Uchimura et al. (2018) reveal additional T-cell-intrinsic functions for NLRC3 in restricting T cell metabolism, T helper 1 and T helper 17 cell responses, and antiviral and autoimmune responses.

Original languageEnglish
Pages (from-to)989-991
Number of pages3
JournalImmunity
Volume49
Issue number6
DOIs
Publication statusPublished - 18 Dec 2018
Externally publishedYes

ASJC Scopus subject areas

  • Immunology and Allergy
  • Immunology
  • Infectious Diseases

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