Protein kinase C{delta} deficiency accelerates neointimal lesions of mouse injured artery involving delayed reendothelialization and vasohibin-1 accumulation

Xue Bai; Andriana Margariti; Yanhua Hu; Yasufumi Sato; Lingfang Zeng; Aleksandar Ivetic; Ouassila Habi; Justin C Mason; Xian Wang; Qingbo Xu

doi:10.1161/ATVBAHA.110.215723

Protein kinase C{delta} deficiency accelerates neointimal lesions of mouse injured artery involving delayed reendothelialization and vasohibin-1 accumulation

Xue Bai, Andriana Margariti, Yanhua Hu, Yasufumi Sato, Lingfang Zeng, Aleksandar Ivetic, Ouassila Habi, Justin C Mason, Xian Wang, Qingbo Xu

Research output: Contribution to journal › Article › peer-review

20 Citations (Scopus)

Abstract

To use protein kinase C (PKC) d-knockout mice to investigate the role of PKCd in lesion development and to understand the underlying mechanism of the vascular disease.

Original language	English
Pages (from-to)	2467-74
Number of pages	8
Journal	Arteriosclerosis Thrombosis and Vascular Biology
Volume	30
Issue number	12
DOIs	https://doi.org/10.1161/ATVBAHA.110.215723
Publication status	Published - 2010

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Bai, X., Margariti, A., Hu, Y., Sato, Y., Zeng, L., Ivetic, A., Habi, O., Mason, J. C., Wang, X., & Xu, Q. (2010). Protein kinase C{delta} deficiency accelerates neointimal lesions of mouse injured artery involving delayed reendothelialization and vasohibin-1 accumulation. Arteriosclerosis Thrombosis and Vascular Biology, 30(12), 2467-74. https://doi.org/10.1161/ATVBAHA.110.215723

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title = "Protein kinase C{delta} deficiency accelerates neointimal lesions of mouse injured artery involving delayed reendothelialization and vasohibin-1 accumulation",

abstract = "To use protein kinase C (PKC) d-knockout mice to investigate the role of PKCd in lesion development and to understand the underlying mechanism of the vascular disease.",

author = "Xue Bai and Andriana Margariti and Yanhua Hu and Yasufumi Sato and Lingfang Zeng and Aleksandar Ivetic and Ouassila Habi and Mason, {Justin C} and Xian Wang and Qingbo Xu",

year = "2010",

doi = "10.1161/ATVBAHA.110.215723",

language = "English",

volume = "30",

pages = "2467--74",

journal = "Arteriosclerosis Thrombosis and Vascular Biology",

issn = "1524-4636",

publisher = "Lippincott Williams & Wilkins",

number = "12",

}

Bai, X, Margariti, A, Hu, Y, Sato, Y, Zeng, L, Ivetic, A, Habi, O, Mason, JC, Wang, X & Xu, Q 2010, 'Protein kinase C{delta} deficiency accelerates neointimal lesions of mouse injured artery involving delayed reendothelialization and vasohibin-1 accumulation', Arteriosclerosis Thrombosis and Vascular Biology, vol. 30, no. 12, pp. 2467-74. https://doi.org/10.1161/ATVBAHA.110.215723

TY - JOUR

T1 - Protein kinase C{delta} deficiency accelerates neointimal lesions of mouse injured artery involving delayed reendothelialization and vasohibin-1 accumulation

AU - Bai, Xue

AU - Margariti, Andriana

AU - Hu, Yanhua

AU - Sato, Yasufumi

AU - Zeng, Lingfang

AU - Ivetic, Aleksandar

AU - Habi, Ouassila

AU - Mason, Justin C

AU - Wang, Xian

AU - Xu, Qingbo

PY - 2010

Y1 - 2010

N2 - To use protein kinase C (PKC) d-knockout mice to investigate the role of PKCd in lesion development and to understand the underlying mechanism of the vascular disease.

AB - To use protein kinase C (PKC) d-knockout mice to investigate the role of PKCd in lesion development and to understand the underlying mechanism of the vascular disease.

U2 - 10.1161/ATVBAHA.110.215723

DO - 10.1161/ATVBAHA.110.215723

M3 - Article

C2 - 20884873

SN - 1524-4636

VL - 30

SP - 2467

EP - 2474

JO - Arteriosclerosis Thrombosis and Vascular Biology

JF - Arteriosclerosis Thrombosis and Vascular Biology

IS - 12

ER -

Protein kinase C{delta} deficiency accelerates neointimal lesions of mouse injured artery involving delayed reendothelialization and vasohibin-1 accumulation

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